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ALS MCQ Answers

ALS MCQ ANSWERS

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100% found this document useful (3 votes)
9K views

ALS MCQ Answers

ALS MCQ ANSWERS

Uploaded by

cameronmct
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd
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ALS MCQ Answers:

Section 1 - ST-segment-elevation myocardial infarction (STEMI):

Q1a. is always accompanied by chest pain.

False

Feedback: The elderly, diabetics and patients with renal disease may not
have chest pain.
Reference: Chapter 4. Diagnosis of acute syndromes

Q1b. rarely causes VF.

False

Feedback: During the acute phase, there is substantial risk of VF.


Reference: Chapter 4. ST-segment-elevation myocardial infarction
(STEMI)

Q1c. may present with ST-depression in leads V1-3 of a 12-lead


ECG.

True

Feedback: A posterior STEMI will show ST-depression in leads V1-3.


Reference: Chapter 4. Figure 4.6

Q1d. may present with new left bundle branch block (LBBB) on
the ECG.

True

Feedback: New LBBB is diagnostic of STEMI.


Reference: Chapter 4. ST-segment-elevation myocardial infarction
(STEMI)
Section 2 - When using transcutaneous pacing:

Q2a. electrical capture typically occurs with a current of 5-10


amps.

False

Feedback: Capture typically occurs with a current of 50-100 mA (i.e. 100


times less).
Reference: Chapter 10. How to perform transcutaneous pacing

Q2b. may be unsuccessful in a patient with hyperkalaemia.

True

Feedback: Hyperkalaemia may prevent successful pacing.


Reference: Chapter 10. How to perform transcutaneous pacing

Q2c. movement artefact may inhibit the pacemaker.

True

Feedback: If there is lot of movement artefact on the ECG, this may be


misinterpreted by the pacemaker and inhibit it.
Reference: Chapter 10. How to perform transcutaneous pacing

Q2d. electrical capture and generation of a QRS complex ensures


return of pulse.

False

Feedback: A QRS complex does not guarantee myocardial contractility.


Absence of pulse in the presence of good electrical capture constitutes
PEA.
Reference: Chapter 10. How to perform transcutaneous pacing
Section 3 - With Regards to the ECG:

Q3a. continuous monitoring via self-adhesive pads is preferable


to using the ECG electrodes.

False

Feedback: Adhesive defibrillator pads should be used in an emergency to


assess the cardiac rhythm.
Reference: Chapter 8. Emergency monitoring

Q3b. if adhesive electrodes are used for 3-lead monitoring, they


should be applied over bone rather than muscle.

True

Feedback: Electrodes should be placed over bone rather than muscle to


minimise interference from muscle artefact.
Reference: Chapter 8. How to attach the monitor

Q3c. the normal PR interval is more than 0.2 s.

False

Feedback: The normal PR interval is between 0.12 and 0.20 s (3-5 small
squares).
Reference: Chapter 8. Heart block: first-degree atrioventricular block

Q3d. the normal QRS complex has a duration < 0.12 s.

True

Feedback: The normal QRS complex interval is <0.12 s (< 3 small


squares).
Reference: Chapter 8. Basic electrocardiography
Section 4 - In drowning:

* Q4a. there is immediate entry of water into the victim’s lungs.

False

Feedback: There is initially laryngospasm and breath holding preventing


entry if water unto the victim's lung.
Reference: Chapter 12. Drowning

Q4b. resuscitation should be considered even it the patient has


been submersed in water for 5 min.

True

Feedback: Submersion durations of less than 10 min are associated with


very high chance of a good outcome, and submersion durations of more
than 25 min are associated with a low chance of good outcome.
Reference: Chapter 12. Drowning: water rescue

Q4c. following submersion, respiratory arrest usually precedes


cardiac arrest

True

Feedback: Cardiac arrest is usually a secondary event following a period


of hypoxia.
Reference: Chapter 12. Drowning: pathophysiology of drowning

Q4d. prophylactics antibiotic therapy should be given routinely.

False

Feedback: Prophylactic antibiotic have not been shown to be of benefit in


preventing chest infection.
Reference: Chapter 12. Drowning: post-resuscitation care after drowning
Section 5 - You arrive at the bedside 4 min after the cardiac
arrest of a 70 kg woman. An IV line is in place but there is no
pulse. The ECG confirms asystole. Two nurses are performing CPR
competently. You would recommend:

Q5a. delivery of a 150 J shock.

False

Feedback: The treatment of asystole does not include defibrillation.


Reference: Chapter 6. Treatment for PEA and asystole

Q5b. sodium bicarbonate 500 mmol IV

False

Feedback: Routine use is not recommended and is associated with a


number of significant side-effects.
Reference: Appendix A. Sodium bicarbonate

Q5c. calcium chloride 5 mL 10% solution IV.

False

Feedback: Indicated only in PEA caused by hyperkalaemia,


hypocalcaemia and overdose of calcium channel blocking drugs.
Reference: Appendix A. Calcium

Q5d. adrenaline 1 mg IV.

True

Feedback: Adrenaline 1 mg IV should be given as soon as intravascular


access is achieved in patients in asystole.
Reference: Chapter 6. Treatment for PEA and asystole
Section 6 - A 55-year-old man on CCU has a witnessed, monitored
VF cardiac arrest. After the 3rd shock, he develops sinus rhythm
with a pulse and starts to breathe spontaneously. He is given
oxygen via a reservoir mask with a flow of 15 L-1. Analysis of
blood gas shows:

Q6a. these suggest that the patient has an acidemia

True

Feedback: A decrease in blood pH below 7.35 indicates an acidaemia.


Reference: Chapter 15. pH

Q6b. these suggest that oxygenation is appropriate for the


inspired concentration.

False

Feedback: The PaO2 should be numerically about 10 less than the


inspired concentration. In this case, this would be > 60 kPa.
Reference: Chapter 15. PaO2

*Q6c. these suggest that the patient has compensatory metabolic


alkalosis.

False

Feedback: There is no compensatory increase in base/bicarbonate. Both


base excess and bicarbonate are reduced indicating a metabolic acidosis.
Reference: Chapter 15. Interpreting the results

*Q6d. these results suggest that 50 mmol of 8.4% sodium


bicarbonate IV is required.
False

Feedback: Not routinely recommended. Has several adverse effects,


including exacerbating intracellular acidosis.
Reference: Appendix A. Sodium bicarbonate

Section 5: -Pulseless electrical activity (PEA)

Q7a. is rarely the first monitored rhythm in a cardiac arrest

False

Feedback: The first monitored rhythm is VF/pVT in only 20% of cardiac


arrests. Therefore, PEA and asystole are relatively more common.
Reference: Chapter 6. Shockable rhythms

Q7b. is characterised by evidence of ventricular activity on the


ECG that would normally be associated with a pulse.

True

Feedback: This is the definition of PEA.


Reference: Chapter 6. Non-shockable rhythms

Q7c. should be treated by giving 300 mg amiodarone IV.

False

Feedback: Amiodarone is only indicated in the treatment of cardiac


arrest due to VF/pVT.
Reference: Chapter 6. Shockable rhythms

Q7d. is usually the cardiac arrest rhythm in patients with severe


hypovolaemia

True

Feedback: Hypovolaemia, usually caused by severe haemorrhage, will


cause PEA.
Reference: Chapter 6. Identification and treatment of reversible cause
Section 8: -With regard to decisions about CPR:

Q8a. the best time to make an anticipatory decision about


whether or not to attempt CPR is when a patient is admitted to
hospital.

False

Feedback: When possible, the best time to make anticipatory decision


about life-sustaining treatments is before the person becomes acutely ill.
Reference: Chapter 16. Deciding whether or not to attempt CPR

Q8b. if you consider that a patient should be 'for CPR', you should
still discuss the decision with the patient.

True

Feedback: Deciding that CPR is appropriate implies a risk of death or


cardiac arrest. Failing to discuss a CPR decision with a patient may deprive
them of the opportunity to refuse CPR if they would not want it.
Reference: Chapter 16. Communication: discussing decisions about COR
with patients

Q8c. if it is agreed that a patient is 'for CPR', there is no need to


document anything in the health records.

False

Feedback: Failing to record decisions and the reasons for making them is
poor clinical practice. Record a clear plan for when the decision should be
reviewed.
Reference: Chapter 16. Recording decisions about CPR

Q8d. if a DNACPR decision is made with a person who has an


implanted cardioverter-defibrillator (ICD), the shock function of
the ICD should then be deactivated.

False
Feedback: Some people with ICDs may not want to receive CPR but
would choose to receive treatment from their ICD, which would be likely to
restore their current clinical situation.
Reference: Chapter 16. Decisions about implanted cardioverter-
defibrillators

Section 9 - Adrenaline

Q9a. has purely alpha-adrenergic effects.

False

Feedback: Adrenaline has both alpha and beta-adrenergic effects.


Reference: Appendix A. Drugs used during cardiac arrest

Q9b. is not associated with any long-term benefits to patients


when given in cardiac arrest.

True

Feedback: Although there is no evidence of long-term benefits from the


use of adrenaline, the improved short-term survival documented in some
studies warrants its continued use.
Reference: Appendix A. Drugs used during cardiac arrest

Q9c. increases systemic vasoconstriction.

True

Feedback: This is due to the alpha-adrenergic effects of adrenaline.


Reference: Appendix A. Drugs used during cardiac arrest

Q9d. improves coronary and cerebral perfusion pressures during


CPR.

True

Feedback: These are some of the alpha-adrenergic effects.


Reference: Appendix A. Drugs used during cardiac arrest
Section 10 - When monitoring the cardiac rhythm:

Q10a. a ventricular rate of between 60-100 beats min-1 is


considered normal.

True

Feedback: The normal heart rate at rest is 60-100 beats min-1.


Reference: Chapter 8. What is the ventricular rate?

Q10b. asystole presents as a completely straight line.

False

Feedback: A completely straight line indicates usually that a monitoring


lead has become disconnected.
Reference: Chapter 8. Is there any electrical activity?

Q10c. at a standard paper speed of 25 mm s-1, the ventricular


rate is calculated by dividing the number of large squares
between consecutive R waves by 60.

False

Feedback: The best way of estimating the heart rate is to count the
number of cardiac cycles (R wave to R wave, including fractions) that
occurs in 6 s (30 large squares)) and multiple by 10. This provides an
estimate of heart rate even when the rhythm is irregular.
Reference: Chapter 8. What is the ventricular rate?

Q10d. A ventricular tachycardia will always require immediate


cardioversion.

False

Feedback: Cardioversion is required only if adverse features (e.g. shock,


syncope, heart failure).
Reference: Chapter 11. Adult tachycardia algorithm
Section 11 - 55-year-old women presents with a 1 h history of
crushing central chest pain, nausea and sweating. Her pulse rate
is 38 min-1, BP 75/45 mmHg. The ECG monitor shows sinus
bradycardia. You should recommend that:

Q11a. atropine 500mcg IV should be given.

True

Feedback: Atropine is the first line treatment in a bradycardia with life-


threatening features in an attempt to increase heart rate and cardiac
output.
Reference: Chapter 11. Adult bradycardia algorithm

Q11b. an adrenaline infusion, 20-100 mcg min-1, may be required.

False

Feedback: An adrenaline infusion may be required, but the correct rate is


2-10 mcg min-1 IV.
Reference: Chapter 11. Adult bradycardia algorithm

* Q11c. opioid analgesia is contraindicated.

False

Feedback: Relief of pain is important, and IV morphine should be titrated


to control symptoms.
Reference: Chapter 4. Immediate treatment

* Q11d. 24% oxygen via a mask should be given until the results
of arterial blood gas analysis are known

False
Feedback: Oxygen by face mask should be given to achieve an oxygen
saturation (SpO2) of 94-98% in the presence of COPD. Initially, this may
require more than 24%.
Reference: Chapter 4. Immediate treatment

Section 12 - During CPR:

Q12a. there is a high risk of transmission of HIV virus from the


patient to the rescuer.

False

Feedback: Transmission of HIV during CPR has never been reported.


Reference: Chapter 5. Sequence for collapsed patient in a hospital

Q12b. personal protective equipment (PPE) should be worn if the


patient has tuberculosis (TB).

True

Feedback: PPE should be used when the victim has serious infection such
as TB or SARS.
Reference: Chapter 5. Sequence for collapsed patient in a hospital

Q12c. wearing latex gloves does not provide sufficient protection


from the electrical current during defibrillation.

True

Feedback: The gloves routinely available do not provide protection from


the electrical current. No part of any person should make contact with the
patient.
Reference: Chapter 9. Safety

* Q12d. if ventilating with high-flow oxygen via an LMA, it must


be disconnected and placed at least 1 m away.

False

Feedback: The ventilation bag can be left connected to the tracheal tube
or supraglottic airway device. No increase in oxygen concentration occurs
in the zone of defibrillation, even with high flow.
Reference: Chapter 9. Safety

Section 13 – Following successful resuscitation from VF cardiac


arrest:

* Q13a. all patients should be given as close to 100% oxygen as


possible.

False

Feedback: If ROSC is achieved, adjust the inspired oxygen to a target


oxygen saturation (SpO2) at 94-98%.
Reference: Chapter 13. Airway and breathing

* Q13b. intubated patient's lungs should be ventilated to achieve


PaCO2 < 4.5 kPa

False

Feedback: Ventilation should be adjusted to achieve normocapnia,


PaCO2 4.7 to 6.0 kPa
Reference: Chapter 13. Airway and breathing

Q13c. continuously monitor the core temperature in patients who


remain comatose after ROSC.

True

Feedback: Continuously monitor the core temperature in patients who


remain comatose after ROSC. Pyrexia is common in the first 2-3 days after
cardiac arrest, and several studies have documented an association
between post-cardiac arrest pyrexia and poor outcomes. Although the
effect of elevated temperature on outcome is not proven, treat any
pyrexia occurring after cardiac arrest with antipyretics or active cooling.
Reference: Chapter 13. Temperature management
Q13d. absence of both pupillary light and corneal reflexes at 72 h
can be used to help predict outcome in comatose patients 72 h
after cardiac arrest.

True

Feedback: This is one of the multimodal categories of tests that can be


used after 72 h.
Reference: Chapter 13. Prognostication

Section 14 – Giving 8.45 sodium bicarbonate

Q14a. may exacerbate an intracellular acidosis.

True

Feedback: It does this by generating CO2 which diffuses intracellular.


Reference: Appendix A. Sodium bicarbonate

* Q14b. is recommended after 5 min of CPR if ROSC has not been


achieved.

False

Feedback: Routine use is not recommended.


Reference: Appendix A. Sodium bicarbonate

Q14c. should be considered as a treatment for arrhythmias due to


tricyclic antidepressant overdose.

True

Feedback: 50 mmol (50 mL of 8.4% solution) can be given with further


doses guided by acid-base status.
Reference: Appendix A. Sodium bicarbonate

Q14d. facilitates release of oxygen to the tissues.

False
Feedback: Shifts the oxygen dissociation curve to the left, inhibiting
release of oxygen to the tissues.
Reference: Appendix A . Sodium bicarbonate

Section 15 – Immediate primary percutaneous coronary


intervention (PPCI)

Q15a. is the preferred treatment for a patient with chest pain


lasting more than 20 min and ST-segment depression in leads V4-
V6 on their ECG.

False

Feedback: ST-depression in V4-V6 is caused by an NSTEMI. There is no


evidence of benefit from immediate reperfusion therapy.
Reference: Chapter 4. Treatment of NSTE ACS (unstable angina and
NSTEMI)

Q15b. is the preferred treatment for a patient with chest pain


lasting more than 20 min and acute ST-segment elevation in leads
V4-V6 on their ECG.

True

Feedback: PPCI is the preferred treatment for STEMI.


Reference: Chapter 4. Treatment of STEMI (or AMI with new LBBB)

Q15c. is the first treatment for unstable angina.

False

Feedback: There is no evidence of benefit from immediate reperfusion


therapy in most patients with NSTE ACS.
Reference: Chapter 4. Treatment of NSTE ACS (unstable angina and
NSTEMI)
Q15d. should be achieved within 120 min of the time of call for
help whenever possible.

True

Feedback: The sooner PPCI is commenced, the more effective it is.


Delays to treatment are associated with high mortality.
Reference: Chapter 4. Treatment of STEMI (or AMI with new LBBB)

Section 16 – the correct management of an adult patient in


ventricular fibrillation includes:

Q16a. digoxin 500 mcg IV.

False

Feedback: Digoxin is not indicated in the treatment of VF. It is used in the


treatment of atrial fibrillation.
Reference: Chapter 6. Treatment of shockable rhythms

Q16b. adrenaline, 1 mg IV after every shock.

False

Feedback: Adrenaline is given after the 3rd shock and subsequently after
alternate shocks (every 3-5 min).
Reference: Chapter 6. Treatment of shockable rhythms

Q16c. atropine 3 mg after 2 loops.

False

Feedback: Atropine is not indicated in the treatment of VF.


Reference: Chapter 6. Treatment of shockable rhythms

Q16d. an initial shock energy of at least 120 J.


True

Feedback: This is the correct energy for the first shock. Subsequent
shocks can be the same or higher energy.
Reference: Chapter 6. Treatment of shockable rhythms

Section 17 – The following statements are correct:

Q17a. adrenaline 1 mg IV should be given to all patients in


cardiac arrest.

False

Feedback: Adrenaline is only given after the 3rd shock in VF. If ROSC is
achieved before this adrenaline will not be required.
Reference: Chapter 6. Treatment of shockable rhythms

Q17b. lidocaine 100 mg is the treatment of choice for all patients


in ventricular tachycardia (VT).

False

Feedback: If the patient has life-threatening features, then cardioversion


will be required. If no adverse features, treatment will depend on the QRS
morphology.
Reference: Chapter 11. Adult tachycardia algorithm

Q17c. adenosine is effective in the treatment of paroxysmal


supraventricular tachycardia.

True

Feedback: This can be tried after vagal manoeuvres if the rhythm is not
atrial flutter.
Reference: Chapter 11. Treatment of regular narrow-complex
tachyarrhythmia

Q17d. the initial dose of amiodarone for shock refractory


ventricular fibrillation is 300 mg IV.

True

Feedback: This is given after the 3rd shock for patients in VF.
Reference: Chapter 6. Treatment of shockable rhythms

Section 18 – Chest Compressions

* Q18a. must not be interrupted to palpate a pulse unless the


patient shows sign of life.

True

Feedback: If an organised rhythm is seen during a 2 min period of CPR,


do not interrupt chest compressions to palpate a pulse unless the patient
shows signs of life suggesting ROSC.
Reference: Chapter 6. Treatment of shockable rhythm

Q18b. are not interrupted for ventilation after tracheal intubation


has occurred.

True

Feedback: As soon as the airway is secured, continue chest


compressions without pausing during ventilation.
Reference: Chapter 6. Maintain high quality, uninterrupted chest
compassions

Q18c. should be performed at a rate if 60 min-1 in adults.

False
Feedback: The correct rate for chest compressions is a rate of 100-120
compressions min-1.
Reference: Chapter 5. Sequence for collapsed patient in a hospital

Q18d. should be started in any unresponsive patient with agonal


breathing.

True

Feedback: Agonal breathing is a sign of cardiac arrest, not a sign of life.


Starting CPR on a patient with low cardiac output is unlikely to be harmful
and may be beneficial.
Reference: Chapter 5. Sequence for collapsed patient in a hospital

Section 19 – In acute severe asthma:

* Q19a. cardiac arrest is secondary to hypercapnia.

False

Feedback: Although the patient may be hypercapnic, cardiac arrest is


secondary to hypoxia.
Reference: Chapter 12. Asthma

* Q19b. oxygen should be titrated to achieve an SpO2 of 88-92%

False

Feedback: Oxygen should be given to achieve an SpO2 of 94-98%. This


may need to be high-flow.
Reference: Chapter 12. Asthma

* Q19c. a PaCO2 of 5.3 kPa is normal.

False

Feedback: Although this is normal for non-asthmatic patient, it is one of


the indicators that the patient has life-treating asthma and becoming
exhausted. PaCO2 is normally low in an asthma attack due to
hyperventilation.
Reference: Chapter 12. Asthma

Q19d. magnesium sulfate 2 g (8 mmol) IV may produce


bronchodilation.

True

Feedback: Magnesium is a bronchodilator and can be effective in these


circumstances.
Reference: Chapter 12. Asthma

Section 20 – in a patient with suspected anaphylaxis:

Q20a. skin and mucosal changes are common features.

True

Feedback: They are often the first feature and present in over 80% of
anaphylactic reactions.

Reference: Chapter 12. Anaphylaxis

Q20b. adrenaline 0.5 mg IM is the first line treatment of choice.

True

Feedback: Adrenaline is the most important drug for the treatment of an


anaphylactic reaction. The initial dose is 0.5 mg IM.
Reference: Chapter 12. Anaphylaxis

Q20c. steroids must be given early.

False
Feedback: They have not been shown to be beneficial when used for
initial resuscitation. They may have a role in the treatment of persisting
asthma-like features.
Reference: Chapter 12. Anaphylaxis

Q20d. colloids are preferred to crystalloids for restoring the


circulation.

False

Feedback: Hartmann's solution or 0.9% saline are suitable fluids for


initial resuscitation. A large volume of fluid may be needed. Consider
colloid infusion as a cause in a patient receiving a colloid at the time of
onset of an anaphylactic reaction.
Reference: Chapter 12. Anaphylaxis

Section 21 - A 65-year-old man with a 2 h history of central chest


pain develops a tachyarrhythmia that appears regular with a rate
of approximately 180 beats min-1. The following treatment should
be given:

* Q21a. if his systolic blood pressure is < 90 mmHg, immediate


cardioversion should be attempted.

True

Feedback: This patient has a life-threatening sign, hypotension, and


therefor DC shock is indicated.
Reference: Chapter 11. Actions to take in all arrhythmias

Q21b. if he has no life-threatening signs, the QRS complex is <


0.12 s and regular, amiodarone 300 mg IV should be given.

False
Feedback: The patient is stable and has narrow-complex tachycardia.
The treatment is vagal manoeuvres and adenosine.
Reference: Chapter 11. Regular narrow-complex tachycardia

Q21c. if he has no life-threatening signs, the QRS complex is <


0.12 s and regular, adenosine 6 mg IV should be given.

True

Feedback: The patient is stable and has a narrow-complex tachycardia.


The treatment is vagal manoeuvres and adenosine.
Reference: Chapter 11. Regular narrow-complex tachycardia

Q21d. if he has no life-threatening signs, the QRS complex is >


0.12 s and regular, amiodarone 300 mg IV should be given.

True

Feedback: This is a regular, broad-complex tachycardia and the initial


treatment is amiodarone.
Reference: Chapter 11. Regular broad-complex tachycardia

Section 22 – Severe hyperkalaemia

Q22a. is defined as a plasma potassium concentration of > 5.5


mmol L-1.

False

Feedback: Hyperkalaemia is defined as a plasma potassium


concentration > 5.5 mmol L-1 severe hyperkalaemia is > 6.5 mmol L-1.
Reference: Chapter 12. Hyperkalaemia

Q22b. causes tall, peaked T waves and ST-depression on the ECG.

True

Feedback: These are characteristics ECG changes.

Reference: Chapter 12. Hyperkalaemia

Q22c. may be caused by renal failure.

True
Feedback: Renal failure (i.e. acute kidney injury or chronic kidney
disease) is a common cause of hyperkalaemia.
Reference: Chapter 12. Hyperkalaemia

Q22d. can be treated by giving 10 mL 1% calcium chloride IV.

Feedback: The correct concentration of calcium chloride is 10%, not 1%.


Reference: Chapter 12. Hyperkalaemia

Section 23 – With regards to decisions about CPR:

Q23a. The only indication for not starting CPR in a patient is the
presence of a recorded valid DNACPR decision.

False

Feedback: There are other reasons for not starting CPR. For example, if it
would not restart heart, CPR should not be attempted.
Reference: Chapter 16. When to withhold CPR

Q23b. any patient is entitled to receive CPR in the event of


cardiac arrest if they insist on it.

False

Feedback: A patient (or their representative) is not entitled to demand


treatment that is clinically inappropriate.
Reference: Chapter 16. Communication: discussing decisions about CPR
with patients

Q23c. overall responsibility for decisions about CPR rests with the
senior clinician in charge of the patient's care.
True

Feedback: The overall responsibility for a decision about CPR, whether


made in advance or at the time of an arrest, rests with the senior health
professional in charge of the person's care at the time of the decision.
Reference: Chapter 16. Deciding whether or not to attempt CPR

* Q23d. if a patient lacks capacity to make a decision about CPR,


their family must be asked to decide whether or not CPR should
be attempted

False

Feedback: If a patient lacks capacity to make decisions, their family


should be involved in those decisions. This is to guide the healthcare team
on what the patient would have chosen had they not lost capacity. It is
important that they do not think that they are entitled to or expected to
make decisions.
Reference: Chapter 16. Communication: discussing decisions about CPR
with those close to patients

Section 24 - In second-degree atrioventricular (AV) heart block:

Q24a. there are always more P waves than QRS complexes

True

Feedback: Second-degree block results in lack of QRS complexes after


some P waves.
Reference: Chapter 8. Heart block: second-degree atrioventricular block

Q24b. the PR interval is always regular.

False

Feedback: There is progressive lengthening of the PR interval in Type I


block.
Reference: Chapter 8. Heart block: second-degree atrioventricular block

Q24c. immediate treatment for a bradycardia will be required.

False
Feedback: Immediate decisions about treatment of these rhythms will be
determined by the effect of the resulting bradycardia on the patient.
Reference: Chapter 8. Heart block: second-degree atrioventricular block

Q24d. when it is Mobitz type II, there is a risk of asystole.

True

Feedback: Mobitz type II is more likely to progress to complex block


asystole.
Reference: Chapter 8. Heart block: second degree atrioventricular block

Section 25 – During advanced life support (ALS)

Q25a. higher defibrillation energies may be required in patients


whose cardiac arrest has been caused by asthma.

True

Feedback: Hyperinflation increases thoracic impedance. Higher energies


should be considered if the first shock fails.
Reference: Chapter 12. Asthma

* Q25b. fine VF must always be treated as asystole.

False

Feedback: If it is not certain whether the ECG shows asystole or very fine
VF, do not spend time attempting to distinguish the rhythm. If the rhythm
appears to be VF give a shock, and if it appears to be asystole continue
chest compressions. Avoid excessive interruptions in chest compressions
for rhythm analysis.
Reference: Chapter 6. Shockable rhythms
Q25c. self-adhesive pads must be placed in the antero-posterior
position in a patient with an implantable cardiover-defibrillator
(ICD).

False

Feedback: Pads should be placed at least 10-15 cm from the ICD or


alternatively in the antero-posterior position.
Reference: Chapter 9. Implanted electronic devices

* Q25d. if the rhythm changes from asystole to VF during the 2


min cycle, a shock should be given.

False

Feedback: CPR should be continued until the end of the 2 min cycle and
then a shock delivered.
Reference: Chapter 6. Non-shockable rhythms (PEA and asystole)

Section 26 – In a patient suspected of having an acute coronary


syndrome (ACS):

Q26a. a single normal 12-lead ECG excludes this as a possible


diagnosis.

False

Feedback: A single normal 12-lead ECG does not exclude ACS.


Reference: Chapter 4. The 12-lead ECG

Q26b. Troponin values above the normal range always indicate


myocardial infarction.

False

Feedback: The release of troponin does not in itself indicate a diagnosis


of ACS. Troponin release aids diagnosis and is a maker of risk when the
history indicates a high probability of AMI. Troponin may be released in
other life-threatening conditions presenting with chest pain such as
pulmonary embolism.
Reference: Chapter 4. Laboratory tests

Q26c. Thrombolysis is contraindicated if the patient has had a


surgical procedure within the last month.

False

Feedback: Major surgery within three weeks is an absolute


contraindication.
Reference: Chapter 4. Fibrinolytic therapy, table 4.2

Q26d. Fibrinolytic therapy is as effective as PPCI.

False

Feedback: Fibrinolytic therapy substantially reduces mortality from AMI


when given during the first few hours after the onset of chest pain but is
less effective than PPCI.
Reference: Chapter 4. Fibrinolytic therapy

Section 27 - A 28-year-old man, known to have asthma, has been


very wheezy for 6 h and has had no relief from his inhalers. On
examination, he is breathless at rest, unable to complete
sentences and has respiratory rate of 36 min-1; there is poor air
entry and wheeze throughout both lung fields. While breathing
oxygen from a reservoir mask (flow 15 L min-1), analysis of an
arterial blood sample shows:

Q27a. oxygenation is lower than predicted from the inspired


concentration.
True

Feedback: High-flow oxygen would provide an FiO2 of around 80%. We


would expect the PaO2 to be 60-70 kPa.
Reference: Chapter 15. PaO2

Q27b. these suggest that the patient has acidaemia.

False

Feedback: The pH > 7.45 is increased indicating an alkalaemia.


Reference: Chapter 15. pH

Q27c. these suggest that the patient has a compensatory


metabolic alkalosis.

True

Feedback: The bicarbonate and base excess are reduced suggesting a


mild metabolic acidosis.
Reference: Chapter 15. Bicarbonate and base excess

Q27d. these suggest that the patient has respiratory alkalosis.

True

Feedback:The pH is increased (> 7.45) indicating an alkalaemia. The


PaCO2 is reduced indicating that there is a respiratory cause for this, a
respiratory alkalosis. There is no increase in bicarbonate or base excess to
suggest this is metabolic in origin.
Reference: Chapter 15. PaCO2

Section 28 - A 35-year-old lady is on the ward following a


cholecystectomy. She complains of abdominal pain and appears
pale and sweaty;

Q28a. a respiratory rate of 30 breaths min-1 may be a sign of


deterioration.

True

Feedback: A high (> 25 min-1) or increasing respiratory rate is a marker


of illness and a warning that the patient may deteriorate suddenly.
Reference: Chapter 3. The ABCDE approach
Q28b. her early warning scores (EWS) may detect evidence of
deterioration.

True

Feedback: To help early detection of critical illness, hospitals use EWS or


calling criteria.
Reference: Chapter 3. The ABCDE approach

Q28c. a normal systolic blood pressure rules out the possibility of


shock.

False

Feedback: In shock, the blood pressure may be normal because


compensatory mechanisms increase peripheral resistance in response to
reduced cardiac output particularly in the younger patients.
Reference: Chapter 3. The ABCDE approach

Q28d. looking for a source of hypovolaemia is the first priority.

False

Feedback: The approach to all deteriorating or critically ill patients is a


complete assessment using an ABCDE approach.

Reference: Chapter 3. The ABCDE approach

Section 29 - The effectiveness of a resuscitation team may be


improved by:

Q29a. early identification of a team leader.

True

Feedback: The team should meet at the beginning of their period on duty
to allocate the team leader. Skill and experience take precedence over
seniority.
Reference: Chapter 2. Resuscitation teams
Q29b. the team leader carrying out all the necessary
interventions.

False

Feedback: The team leader provides guidance, direction and instruction


to the team members to enable successful completion of their stated
objective. They lead by example and integrity.
Reference: Chapter 2. Team working including leadership

Q29c. the team should meet at the beginning of their period on


duty to identify everyone's skills and experience and allocate
roles.

True

Feedback: The team should meet at the beginning of their period on duty
to identify everyone's skills and experience and allocate roles.
Reference: Chapter 2. Resuscitation teams

Q29d. ensuring that the most senior person acts as the team
leader.

False

Feedback: Skill and experience take precedence over seniority.


Reference: Chapter 2. Resuscitation teams

Section 30 - Following successful resuscitation from a cardiac


arrest:

Q30a. the patient may be hyperkalaemic.

True
Feedback: Immediately after a cardiac arrest, there is typically a period
of hyperkalaemia.
Reference: Chapter 13. Optimising organ function

Q30b. maintain the patient’s blood glucose between 4.0-8.0 mmol


L-1.

False

Feedback: Based on the available data and expect consensus, following


ROSC, maintain blood glucose at < 10 mmol L-1 and avoid hypoglycaemia
(< 4.0 mmol L-1).
Reference: Chapter 13. Brain: optimising neurological recover

Q30c. cerebral perfusion returns to normal immediately with


ROSC.

False

Feedback: Immediately after ROSC, there is a period of cerebral


hyperaemia due to impaired autoregulation.
Reference: Chapter 13. Brain: optimising neurological recovery

Q30d. seizures occurs in > 50% of patients who remain comatose.

False

Feedback: Seizures occur in about one-third of patients who remain


comatose after ROSC.
Reference: Chapter 13. Brain: optimising neurological recovery

Section 31 – With reference to the rhythm strip:


Q31a. sinus bradycardia is present.

False

Feedback: The best way to estimate the heart rate is to count the
number of cardiac cycles (R wave to R wave, including fractions) that
occur in 6 s (30 large squares) and multiply by 10. There are 6.7 cardiac
cycles in 6 s. Therefore, there ventricular rate is 67 min-1.

Q31b. Primary PCI may be indicated.

True

Feedback: This ECG is consistent with a STEMI and PPCI is the preferred
method of reperfusion.

Q31c. cardiac monitoring is advisable.

True

Feedback: All critically ill patient should have ECG monitoring as soon as
possible.

Q31d. aspirin should be avoided.

False

Feedback: Immediate general treatment for ACS includes aspirin 300 mg


crushed or chewed as soon as possible.

Section 32 - With reference to the rhythm strip:


Q32a. this rhythm can be associated with a spontaneous
circulation.

False

Feedback: The rhythm is ventricular fibrillation and not associated with a


detectable cardiac output.
Reference: Chapter 8. How to read a rhythm strip

* Q32b. adrenaline 1 mg IV is the initial treatment of choice.

False

Feedback: Adrenaline is only indicated after 3rd shock in VF.


Reference: Chapter 6. Shockable rhythms (VF/pVT)

* Q32c. a precordial thump may be indicated.

True

Feedback: A precordial thump has a very low success rate for


cardioversion of a shockable rhythm. Its routine use is not recommended.
Consider a precordial thump only when it can be used without delay whilst
awaiting the arrival of a defibrillator in a monitored VF/pVT arrest.
Reference: Chapter 8. Precordial thump

* Q32d. defibrillation is the treatment of choice in the pulseless


patient.

True

Feedback: Early defibrillation is one of the few interventions that


contribute to improved survival from VF.
Reference: Chapter 6. Introduction

Section 33 - With reference to the rhythm strip:


Q33a. the ventricular rate is in the range of 90-110 min-1.

False

Feedback: The best way of estimating the heart is to count the number
of cardiac cycles (R wave to R wave, including fractions) that occur in 6 s
(30 large squares) and multiple by 10. The irregular rhythm causes slight
variation in the rate. There are approximately 20 cardiac cycles in 6 s.
Therefore, the ventricular rate is approximately 200 min-1.
Reference: Chapter 8. How to read a rhythm strip

Q33b. the rhythm is irregular.

True

Feedback: The R-R interval is not constant making the rhythm irregular.
Reference: Chapter 8. How to read a rhythm strip

Q33c. the rhythm is supraventricular in origin.

True Feedback: The QRS complex is < 0.12 s (3 small squares) and
therefore originates above the ventricles.
Reference: Chapter 8. How to read a rhythm strip

Q33d. the rhythm is atrial flutter.

False

Feedback: The R-R intervals are totally irregular and the QRS complex is
of constant morphology, so the rhythm is atrial fibrillation.
Reference: Chapter 8. How to read a rhythm strip

Section 34 - With reference to the rhythm strip:


* Q34a. the ventricular rate is in the range 150-200 min-1.

False

Feedback: The best way of estimating the heart rate is to count the
number of cardiac cycles (R wave to R wave, including fractions) that
occur in 6 s (30 large squares) and multiply by 10. There are 11.5 cardiac
cycles in 6 s, therefore, the ventricular rate is 115 min-1. Alternatively, the
number of cardiac cycles occurring in 3 s is 5.7, giving a rate of 114 min-
1.
Reference: Chapter 8. How to read a rhythm strip

Q34b. the rhythm is regular.

True

Feedback: The R-R intervals are constant; therefore, the rhythm is


regular.
Reference: Chapter 8. How to read a rhythm strip

* Q34c. the rhythm originates in the ventricles.

False

Feedback:
The QRS complex is < 0.12 s (3 small squares) and therefore
originates above the ventricles.

Reference: Chapter 8. How to read a rhythm strip

Q34d. P waves are clearly present.

False

Feedback: No P waves are visible, only QRS and T waves.


Reference: Chapter 8. How to read a rhythm strip

Section 35 - With reference to the rhythm strip:


Q35a. the ventricular rate is in the range of 25-35 min-1.

True

Feedback: The best way of estimating the heart rate is to count the
number of cardiac cycles (R wave to R wave, including fractions) that
occur in 6 s (30 large squares) and multiple by 10. The number of cardiac
cycles occurring n 6 s is 2.8. Therefore, the ventricular rate is 28 min-1.
Reference: Chapter 8. How to read a rhythm strip

Q35b. atropine may be indicated.

True

Feedback: If adverse features are present, atropine 500 mcg IV is the


initial treatment.
Reference: Chapter 11. Bradyarrhythmia

Q35c. the atrial rate is in the range 40-50 min-1.

False

Feedback: The atrial rate can be estimated by counting the number if


'atrial cycles' (P wave to P wave, including fractions) that occur in 6 s (30
large squares) and multiplying by 10. The number of atrial cycles
occurring in 6 s in 6.2. Therefore, the atrial rate is 62 min-1.
Reference: Chapter 8. How to read a rhythm strip

Q35d. the duration of the QRS complex is prolonged.

True

Feedback: The QRS complex is > 0.12 s, 3 small squares.


Reference: Chapter 8. How to read a rhythm strip

Section 36 - With reference to the rhythm strip:


Q36a. this rhythm is always associated with unconsciousness.

False

Feedback: The patient may be conscious or have other life-threatening


signs suck shock or heart failure.
Reference: Chapter 11. Bradyarrhythmia

Q36b. a patient with this rhythm will not have a pulse.

False

Feedback: This rhythm is not always associated with cardiac arrest; the
patient may have shock or heart failure.
Reference: Chapter 11. Bradyarrhythmia

Q36c. if the patient is dyspnoeic and hypotensive, systolic blood


pressure 80 mmHg, transvenous pacing may be appropriate.

True

Feedback: These are life-threatening signs and if there is no response to


drugs, transvenous pacing is appropriate.
Reference: Chapter 11. Bradyarrhythmia

Q36d. the patient is at risk of developing asystole.

True

Feedback: This is patient has complete heart block with broad complexes
which puts them at risk of asystole.
Reference: Chapter 11. Bradyarrhythmia

Section 37 - With reference to the rhythm strip:


Q37a. the ventricular rate is in the range 50-60 min-1.

False

Feedback: The best way of estimating the heart rate is to count the
number of cardiac cycles (R wave to R wave, including fractions) that
occur in 6 s (30 large squares) and multiply by 10. The number of cardiac
cycles occurring in 6 s is 2.5. Therefore, the ventricular rate is 25 min-1.
Reference: Chapter 8. How to read a rhythm strip

* Q37b. the PR interval is normal.

False

Feedback: The PR interval is 0.28 s (7 small squares). This is prolonged


and represents first-degree heart block.
Reference: Chapter 8. Heart block: first-degree atrioventricular block

Q37c. atropine 0.5 mg IV is the initial treatment of this rhythm in


a patient with adverse signs.

True

Feedback: Atropine 0.5 mg IV up to a maximum of 3 mg is the initial


treatment of a bradycardia in a patient with adverse features.
Reference: Chapter 11. Bradyarrhythmia

Q37d. adrenaline is contraindicated in the presence of this


rhythm.

False

Feedback: Adrenaline may be used if pacing cannot be achieved


promptly.
Reference: Chapter 11. Bradyarrhythmia

Section 38 - With reference to the rhythm strip:


Q38a. the ventricular rate is greater than 200 min-1.

True

Feedback: The best way to estimating the heart rate is to count the
number if cardiac cycles (R wave to R wave, including fractions) that occur
in 6 s (30 large squares) and multiply by 10. The number of cardiac cycles
occurring in 6 s is 21.8. Therefore, the ventricular rate is 218 min-1.
Reference: Chapter 8. How to read a rhythm strip

Q38b. the rhythm is irregular.

False

Feedback: The interval between complexes is regular.


Reference: Chapter 8. How to read a rhythm strip

Q38c. the QRS complex is abnormal.

True

Feedback:The complexes are broad, greater than 0.12 s and are not
normal morphology.
Reference: Chapter 8. How to read a rhythm strip

Q38d. P waves are clearly visible.

False

Feedback: No P waves are visible.


Reference: Chapter 8. How to read a rhythm strip

Section 39 - With reference to the rhythm strip:


Q39a. atropine may be appropriate treatment for this rhythm.

False

Feedback: Atropine is not indicated in the treatment of tachycardia.


Reference: Chapter 11. Tachyarrhythmia, figure 11.1

Q39b. if the patient is conscious, this rhythm does not require


any treatment.

False

Feedback: This is a regular, broad-complex tachycardia, and may


progress to VF. Treatment with amiodarone IV will be required.
Reference: Chapter 11. Regular broad-complex tachycardia

Q39c. in the presence of a systolic blood pressure of 70 mmHg, a


synchronised cardioversion is the treatment of choice.

True

Feedback: A systolic blood pressure of 70 mmHg is an adverse feature


and therefore synnchronised cardioversion is the treatment of choice.
Reference: Chapter 11. Tachyarrhythmia

Q39d. amiodarone may be indicated.

True

Feedback: In the absence of adverse features, it may be treated with


amiodarone, 300 mg IV over 20-60 min.
Reference: Chapter 11. Tachyarrhythmia

Section 40 - With reference to rhythm strip:


Q40a. the ventricular rate is in the range 60-80 min-1.

False

Feedback: There are no QRS complexes this is ventricular standstill.


Reference: Chapter 8. How to read a rhythm strip

Q40b. the rhythm is complete heart block.

False

Feedback: In third-degree heart block, there are both P waves and QRS
complexes, but no relationship between then. Only P waves visible.
Reference: Chapter 8. Heart block: third-degree atrioventricular block

Q40c. external pacing may be indicated.

True

Feedback: Pacing in the presence of atrial activity but no QRS complexes


is more likely to achieve a cardiac output than most cases of complete
asystole.
Reference: Chapter 8. How to read a rhythm strip

Q40d. amiodarone 300 mg IV should be given.

False

Feedback: Amiodarone is not indicated in ventricular standstill.


Reference: Appendix A. Amiodarone

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